Cell Senescence Entries for AKT1
- Cell Types
- Aortic endothelial, Dermal microvascular endothelial cells, Umbilical vein endothelial
- Cell Lines
- Primary cell
- Cancer Cell?
- No
- Method
- Overexpression
- Type of senescence
- Oncogene-induced
- Senescence Effect
- Induces
- Observations
- Expression of constitutively activated AKT1 in endothelial cells promoted senescence-like growth arrest with the cells displaying enlarged, flattened morphology and increased SA-beta-galactosidase activity. This senescence effect was dependent on the p53/p21 pathway.
- Primary Reference
AKT1 Gene Information
- HGNC symbol
- AKT1
- Aliases
- AKT; PKB; RAC; CWS6; PRKBA; PKB-ALPHA; RAC-ALPHA
- Common name
- AKT serine/threonine kinase 1
- Entrez Id
- 207
- Description
- The serine-threonine protein kinase encoded by the AKT1 gene is catalytically inactive in serum-starved primary and immortalized fibroblasts. AKT1 and the related AKT2 are activated by platelet-derived growth factor. The activation is rapid and specific, and it is abrogated by mutations in the pleckstrin homology domain of AKT1. It was shown that the activation occurs through phosphatidylinositol 3-kinase. In the developing nervous system AKT is a critical mediator of growth factor-induced neuronal survival. Survival factors can suppress apoptosis in a transcription-independent manner by activating the serine/threonine kinase AKT1, which then phosphorylates and inactivates components of the apoptotic machinery. Mutations in this gene have been associated with the Proteus syndrome. Multiple alternatively spliced transcript variants have been found for this gene. [provided by RefSeq, Jul 2011]
AKT1 Ontologies
- Gene Ontology
-
Process: GO:7186; G-protein coupled receptor signaling pathway
GO:82; G1/S transition of mitotic cell cycle
GO:7249; I-kappaB kinase/NF-kappaB signaling
GO:38061; NIK/NF-kappaB signaling
GO:31295; T cell costimulation
GO:31929; TOR signaling
GO:6924; activation-induced cell death of T cells
GO:7568; aging
GO:43276; anoikis
GO:8637; apoptotic mitochondrial changes
GO:30154; cell differentiation
And 120 more GO terms Cellular component: GO:5911; cell-cell junction
GO:36064; ciliary basal body
GO:5737; cytoplasm
GO:5829; cytosol
GO:15630; microtubule cytoskeleton
GO:5739; mitochondrion
GO:5654; nucleoplasm
GO:5634; nucleus
GO:5886; plasma membrane
GO:98794; postsynapse
GO:43234; protein complex
And 3 more GO terms Function: GO:71889; 14-3-3 protein binding
GO:5524; ATP binding
GO:32794; GTPase activating protein binding
GO:19899; enzyme binding
GO:42802; identical protein binding
GO:16301; kinase activity
GO:30235; nitric-oxide synthase regulator activity
GO:5547; phosphatidylinositol-3,4,5-trisphosphate binding
GO:43325; phosphatidylinositol-3,4-bisphosphate binding
GO:5515; protein binding
GO:5080; protein kinase C binding
And 5 more GO terms
Homologs of AKT1 in Model Organisms
- Caenorhabditis elegans
- akt-2
- Caenorhabditis elegans
- akt-1
- Danio rerio
- AKT1
- Drosophila melanogaster
- Akt1
- Mus musculus
- Akt1
- Rattus norvegicus
- Akt1
- Saccharomyces cerevisiae
- SCH9
- Saccharomyces cerevisiae
- YPK1
- Saccharomyces cerevisiae
- YPK2
In other databases
- GenAge model organism genes
- A homolog of this gene for Caenorhabditis elegans is present as akt-1
- A homolog of this gene for Saccharomyces cerevisiae is present as SCH9
- A homolog of this gene for Drosophila melanogaster is present as Akt1
- A homolog of this gene for Saccharomyces cerevisiae is present as YPK1
- A homolog of this gene for Mus musculus is present as Akt1
- GenAge human genes
- This gene is present as AKT1
- GenDR gene manipulations
- A homolog of this gene for Saccharomyces cerevisiae is present as SCH9
- LongevityMap
- This gene is present as AKT1
External links
- OMIM
- 164730
- Ensembl
- ENSG00000142208
- Entrez Gene
- 207
- UniGene
- 525622
- 1000 Genomes
- 1000 Genomes
- HPRD
- GenAtlas
- AKT1
- GeneCards
- AKT1