- Symbol
- Trp53
- Aliases
- bbl; bfy; bhy; p44; p53; Tp53
- Name
- Transformation related protein 53
- Organism
- Mus musculus (AnAge)
- Known functions and activities
- Tumour supressor involved in cell cycle regulation, apoptosis, and DNA repair
- Observations
- Contrary to p53-null mice, mice with a p53 mutation (Ser18), affecting the substrate site for ATM and ATM-related (ATR) protein kinases, do not display an increased mortality due to early-onset tumors. Survival is however compromised, though not as severely as in the case of p53-null mice, and they still spontaneously develop late-onset tumors. Mutants live significantly shorter compared with wild-type mice (up to 23% less).
- % change in avg or median lifespan
- Median lifespan is up to 23% lower.
- Lifespan Effect
- Decrease
- Genetic Manipulation
- Mutation
- Longevity Category
- Pro-Longevity
- Primary reference
-
- Observations
- Mutant mice with activated Trp53 display enhanced resistance to spontaneous tumours and signs of premature ageing including reduced lifespan, osteoporosis, organ atrophy and a diminished stress tolerance.
- Lifespan Effect
- Decrease
- Genetic Manipulation
- Mutation
- Longevity Category
- Pro-Longevity
- Primary reference
-
- Observations
- super p53' mice do not show any indication of premature aging, Mice carrying p53-tg alleles in addition to the two endogenous alleles, exhibit an enhanced response to DNA damage and are significantly protected from cancer when compared with normal mice. This is probably reflecting the fact that p53 is under normal regulatory control.
- Lifespan Effect
- Not Recorded
- Genetic Manipulation
- Overexpression
- Primary reference
-
- Observations
- Haploid loss of p53 overcame embryonic senescence but failed to prevent the adult mutant mice from prematurely aging, which included decreased life span, reduced body fat deposition, osteoporosis, skin atrophy, and decreased wound healing.
- Lifespan Effect
- Decrease
- Genetic Manipulation
- Deletion
- Longevity Category
- Pro-Longevity
- Primary reference
-
- Observations
- Overexpression of p44 upsets the balance that normally exists between the full-length and short forms of p53 and leads to a phenotype of growth suppression and premature aging in mice (40% reduction in MLS). Growth suppression by p44 links small size, proliferation deficits, cellular senescence, and organismal aging to abnormal IGF signaling in the mouse.
- % change in max lifespan
- Maximum lifespan is 40% lower.
- Lifespan Effect
- Decrease
- Genetic Manipulation
- Overexpression of the short isoform of p53 (p44)
- Primary reference
-
- Observations
- Mice lacking both p27 and p53 exhibit a decreased lifespan and develop unique tumors, including papillary carcinoma of the colon, hemangiosarcoma, and leiomyosarcoma.
- Lifespan Effect
- Decrease
- Genetic Manipulation
- Deletion
- Longevity Category
- Pro-Longevity
- Primary reference
-
- GenAge human genes
- A homolog of this gene for Homo sapiens is present as TP53
- LongevityMap
- A homolog of this gene for Homo sapiens is present as TP53
- CellAge
- A homolog of this gene for Homo sapiens is present as TP53
- Danio rerio
- tp53
- Homo sapiens
- TP53
- Rattus norvegicus
- Tp53
- Mouse Genome Informatics
- Search
- Ensembl
- Search
- Entrez Gene
- View on Entrez Gene database (22059)
- Homologues
- Search HomoloGene
- UniProt
- Search
- Internet
- Search Google or Search Google Scholar